[1] MILLER K D, NOGUEIRA L, MARIOTTO A B, et al. Cancer treatment and survivorship statistics, 2019[J]. CA Cancer J Clin, 2019, 69(5):363-385.
[2] HAGGAR F A, BOUSHEY R P.Colorectal cancer epidemiology:incidence, mortality, survival, and risk factors[J]. Clin Colon Rect Surg, 2009, 22(4):191-197.
[3] WENG M, WU D, YANG C, et al. Noncoding RNAs in the development, diagnosis, and prognosis of colorectal cancer[J]. Transl Res, 2017, 181:108-120.
[4] 王希君, 沈容芳, 王想, 等.甲胎蛋白高表达和低表达肝细胞肝癌的基因表达谱差异分析[J]. 中华肿瘤杂志, 2020, 42(5):396-402.
[5] 安澜, 曾红梅, 郑荣寿, 等.2015年中国肝癌流行情况分析[J]. 中华肿瘤杂志, 2019, 41(10):721-727.
[6] GELFMAN S, WANG Q, MCSWEENEY K M, et al. Annotating pathogenic non-coding variants in genic regions[J]. Nat Commun, 2017, 8(1):236.
[7] YU S T, ZHONG Q, CHEN R H, et al. CRLF1 promotes malignant phenotypes of papillary thyroid carcinoma by activating the MAPK/ERK and PI3K/AKT pathways[J]. Cell Death Dis, 2018, 9(3):371.
[8] TORMO A J, MELIANI Y, BEAUPRÉ L A, et al. The composite cytokine p28/cytokine-like factor 1 sustains B cell proliferation and promotes plasma cell differentiation[J]. J Immunol, 2013, 191(4):1657-1665.
[9] DAGONEAU N, BELLAIS S, BLANCHET P, et al. Mutations in cytokine receptor-like factor 1(CRLF1) account for both Crisponi and cold-induced sweating syndromes[J]. Am J Hum Genet, 2007, 80(5):966-970.
[10] Kass D J, YU G, LOH K S, et al. Cytokine-like factor 1 gene expression is enriched in idiopathic pulmonary fibrosis and drives the accumulation of CD4+ T cells in murine lungs:evidence for an antifibrotic role in bleomycin injury[J]. Am J Pathol, 2012, 180(5):1963-1978.
[11] HUARTE M, GUTTMAN M, FELDSER D, et al. A large intergenic noncoding RNA induced by p53 mediates global gene repression in the p53 response[J]. Cell, 2010, 142(3):409-419.
[12] BASAK D, UDDIN M N, HANCOCK J.The Role of Oxidative Stress and Its Counteractive Utility in Colorectal Cancer (CRC)[J]. Cancers (Basel), 2020, 12(11):3336.
[13] KOVEITYPOUR Z, PANAHI F, VAKILIAN M, et al. Signaling pathways involved in colorectal cancer progression[J]. Cell Biosci, 2019, 9:97.
[14] LI Q, DAI Y, WANG F, et al. Differentially expressed long non-coding RNAs and the prognostic potential in colorectal cancer[J]. Neoplasma, 2016, 63(6):977-983.
[15] PAN S, LIU Y, LIU Q, et al. HOTAIR/miR-326/FUT6 axis facilitates colorectal cancer progression through regulating fucosylation of CD44 via PI3K/AKT/mTOR pathway[J]. BBA-Mol Cell Res, 2019, 1866(5):750-760.
[16] SOLEIMANI A, RAHMANI F, FERNS G A, et al. Role of Regulatory oncogenic or tumor suppressor miRNAs of PI3K/AKT signaling axis in the pathogenesis of colorectal cancer[J]. Curr Pharm Design, 2018, 24(39):4605-4610.
[17] BAHRAMI A, KHAZAEI M, HASANZADEH M, et al. Therapeutic potential of targeting PI3K/AKT pathway in treatment of colorectal cancer:rational and progress[J]. J Cell Biochem, 2018, 119(3):2460-2469.
[18] SONG M, BODE A M, DONG Z, et al. AKT as a therapeutic target for cancer[J]. Cancer Res, 2019, 79(6):1019-1031.
[19] POLIVKA J J, JANKU F.Molecular targets for cancer therapy in the PI3K/AKT/mTOR pathway[J]. Pharmacol Therapeut, 2014, 142(2):164-175. |